atrial natriuretic peptide , ANP (skrót)

Plasma atrial natriuretic peptide was extracted according to Sagnella et al.

Atrial natriuretic peptide causes the distal convoluted tubule to excrete more sodium.

Atrial natriuretic peptide is released by the heart in response to high blood pressure and high salinity of the blood.

"Atrial natriuretic peptide attracts human spermatozoa in vitro".

In the kidney it is inhibited by atrial natriuretic peptide, causing natriuresis and diuresis.

Atrial natriuretic peptide forms a parallel negative feedback loop in an endocrinological contrast to the renin-angiotensin system.

Three types of atrial natriuretic peptide receptors have been identified on which natriuretic peptides act.

The measurement of plasma atrial natriuretic peptide indicated that these changes were due to the gravitational effects on the distensibility of the right heart.

Atrial natriuretic peptide, a peptide hormone.

One such compensatory factor is an increase in various hormone levels, including renin, aldosterone, norepinephrine, atrial natriuretic peptide, and prostaglandins.

It is stimulated by angiotensin II and aldosterone, and inhibited by atrial natriuretic peptide.

The median preoptic nucleus is highly involved in cardiovascular regulation, including the release of atrial natriuretic peptide by the heart in response to high blood volume.

Body water is regulated by hormones, including anti-diuretic hormone (ADH), aldosterone and atrial natriuretic peptide.

Atrial natriuretic peptide (CDD/ANP-99-126) is a hormone system of clinical importance.

Moreover, our patients showed normal plasma values of glucagon and atrial natriuretic peptides, the only other hormones known to influence extracellular cyclic nucleotide values.

Dysregulation of dopaminergic neurotransmission and Atrial natriuretic peptide homeostasis resulting from epigenetic mechanisms has been implicated in various eating disorders.

Responses can be endocrine, autonomic or behavioral, and responses to spikes in blood sodium levels include the release of atrial natriuretic peptide and oxytocin.

In 1980, this work led to his discovery and isolation of atrial natriuretic peptide (ANP), a polypeptide hormone secreted by heart muscle cells.

The lower limits of sensitivity for glucagon and atrial natriuretic peptide assay were 3.7 pmol/l and 1.1 pmol/l, respectively.

A atrial natriuretic peptide receptor is a receptor for atrial natriuretic peptide.

In addition, stretching of atrial receptors increases secretion of atrial natriuretic peptide (ANP), which promotes increased water and sodium excretion through the urine.

Various endocrine hormones coordinate these endocrine functions; these include renin, angiotensin II, aldosterone, antidiuretic hormone, and atrial natriuretic peptide, among others.

Atrial Natriuretic Peptide in Plasma of Volume-Overloaded Children with Chronic Renal Failure (co-author, 1985)

Functionally, its importance can be understood because lesions to the median preoptic nucleus generally cause inappropriate fluid composition, water intake and release of atrial natriuretic peptide (ANP).

Current research has linked RE1/NRSE activity with the regulation of the expression of the atrial natriuretic peptide (ANP) gene.