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Stasis of urine in post-renal azotemia has the same effect.
Uremic pericarditis is correlated to the degree of azotemia in the system.
Clinically, the most significant concern is the development of azotemia secondary to reduced renal perfusion.
Nephron loss secondary to ischemic injury also contributes to the development of azotemia in these patients.
He also had developed transient oliguria (no azotemia) during treatment with supplemental fluid therapy.
When kidneys fail to filter properly, waste accumulates in the blood and the body, a condition called azotemia.
Prerenal azotemia is caused by a decrease in blood flow (hypoperfusion) to the kidneys.
As a result diuresis of these patients will result in hypovolemia and pre-renal azotemia.
Uremia is the pathological manifestations of severe azotemia.
It is also seen in some patients with kidney failure and azotemia, and in carbon dioxide toxicity.
Electrolyte imbalance and azotemia may be responsible for the decrease in acetylcholine activity in the brain.
Stage 3 the patient develops shock with azotemia and acid-base disturbances; has significant coagulation abnormalities.
Postrenal azotemia and hydronephrosis can be observed following the obstruction of urine flow through one or both ureters.
Metabolic abnormalities associated with uroperitoneum include azotemia, hyperkalemia, hyponatremia and hypochloremia.
Azotemia, hyperuricemia, and hyperphosphatemia are also associated with TLC.
Investigation revealed Gram positive diplococci in her sputum, neutrophilia, mild anemia, and azotemia.
Proteinuria, urinary casts, azotemia, and metastatic calcification (especially in the kidneys) may develop.
Renal Function events: 1 case of azotemia, 1 case of renal failure.
Renal azotemia (as for urea)
Oliguria and azotemia develop in severe cases as a result of renal involvement due to hypoxia and toxemia.
The BUN:Cr in prerenal azotemia is greater than 20.
PCV and total protein are usually both increased due to fluid loss, and the horse displays a prerenal azotemia.
Prerenal causes of AKI ("pre-renal azotemia") are those that decrease effective blood flow to the kidney.
Francis et al [ 12] reported that abnormal sodium levels, fever/hypothermia and azotemia were independent predictors for delirium in elderly patients.
During the check-up moderate azotemia, mild erythronormoblastic anemia, proteinuria and lowering of left vascular ejection fraction were also identified.