infarcted

It is caused by infarcted muscle tissue, usually in the thigh.

It is used to determine areas of reversible ischemia and infarcted tissue in the heart.

In practice, this may be an area of ischaemic or infarcted myocardium, or underlying scar tissue.

The resulting two sets of images are compared with each other to distinguish ischemic from infarcted areas of the myocardium.

Since the electrical characteristics of the infarcted tissue change (see pathophysiology section), arrhythmias are a frequent complication.

Also MiR-1 is downregulated in myocardial infarcted tissue compared to healthy heart tissue.

Studying an other EKG, Jason had the feeling that the infarcted area of the heart was expanding.

The AT receptor may play role in the generation of reperfusion arrhythmias following restoration of blood flow to ischemic or infarcted myocardium.

Disopyramide decreases the inhomogeneity between infarcted and normal myocardium refractory periods; in addition to lengthening the refractory period.

However, studies have shown NRG-1 promotes myocardial regeneration through hyperplasia, and prevents hypertrophy surrounding infarcted areas (cite).

Type II is an erosion of the infarcted myocardium, which is suggestive of a slow tear of the dead myocardium.

The shear stress between the infarcted segment and the surrounding normal myocardium (which may be hypercontractile in the post-infarction period) makes it a nidus for rupture.

One NSAID in particular, indomethacin can inhibit new collagen deposition thus impairing the healing process for the infarcted region.

The interstitium at the margin of the infarcted area is initially infiltrated with neutrophils, then with lymphocytes and macrophages, who phagocytose ("eat") the myocyte debris.

Regional expression of endothelin-1, ANP, IGF-1, and LV wall stress in the infarcted rat heart.

Infarct expansion, or thinning and lengthening of the infarcted myocardium, is seen within hours after myocardial necrosis and is usually complete within three weeks after infarction.

This process also occurs in animals under hypoxic (or partially anaerobic) conditions, found, for example, in overworked muscles that are starved of oxygen, or in infarcted heart muscle cells.

With SPECT, interior and posterior abnormalities and small areas of infarction can be identified, as well as the occluded blood vessels and the mass of infarcted and viable myocardium.

For example, Carpenter et al. demonstrated efficient cardiac differentiation of human iPS cells that gave rise to progenitors that were retained within the infarcted rat heart, and reduced remodeling of the heart after ischemic damage.

Scintigraphy may recognise oesteonecrosis somewhat earlier, but is rather uncertain because of poor anatomic resolution, false negative examinations with bilateral disease, and because the scintigraphic activity in the infarcted bone varies with the stage of the bone disease.

In another study with a mouse model of acute myocardial infarction, miR-21 expression was found to be significantly lower in infarcted areas and overexpression of miR-21 in those mice via adenovirus-mediated gene transfer decreased myocardial infarct size.