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These proteases rely upon binding of lipopolysaccharide for activity.
It is a constituent of the lipopolysaccharide.
It is 0.2 - 1.0 μm and lacks a lipopolysaccharide biosynthetic machinery.
Lipopolysaccharide is a component of the cell wall of Gram-negative bacteria.
Further reactions join the O-antigen to the core polysaccharide to form the full lipopolysaccharide.
First the "O" antigen type is determined based on polysaccharides associated with lipopolysaccharide.
Species in this group lack lipopolysaccharide.
The lipopolysaccharide character of enteric endotoxins was elucidated in the 1944 by Shear.
CD22 is normally up-regulated by lipopolysaccharide binding to Toll-like receptors.
It has several flagellae, inserted at either pole of the cell, and a lipopolysaccharide cell wall.
In contrast to Bacteroides spp., Fusobacteria have a potent lipopolysaccharide.
It detects lipopolysaccharide from Gram-negative bacteria and is thus important in the activation of the innate immune system.
This enzyme participates in lipopolysaccharide biosynthesis.
A recent study compared the effectiveness of melatonin and other similar compounds on the lipopolysaccharide induced lipid peroxidation.
Pinoline was also shown to be more effective than Vitamin E at reducing lipopolysaccharide activity in the retina.
A Gram-negative cell will lose its outer lipopolysaccharide membrane, and the inner peptidoglycan layer is left exposed.
This reaction inactivates the lipopolysaccharide (endotoxin).
The Parachlamydiaceae are not recognized by monoclonal antibodies that detect Chlamydiaceae lipopolysaccharide.
Lipopolysaccharide (LPS) is a prototypical example of an endotoxin.
The protein encoded by this gene is involved in binding lipopolysaccharide with TLR 4.
All of the collagen-immunized mice had swollen and red paws and ankle joints within 3 days after lipopolysaccharide injection.
Recent study has shown that CD14, a lipopolysaccharide receptor, is itself involved in the stimulation of cell proliferation [ 21].
A methanol extract (100-400 mg/kg) was found to protect against airway inflammation in response to lipopolysaccharide treatment of mice.
Lipopolysaccharide is produced by gram-negative bacteria and stimulates the production of free radicals which in turn cause lipid peroxidation.
The outer leaflet of the membrane comprises a complex lipopolysaccharide whose lipid portion acts as an endotoxin.