non-shivering
nonshivering

Other forms of 'non-shivering thermogenesis' may be used by human populations that encounter cold.

A fun little non-security and also non-shivering tidbit just popped up.

However, brown adipose tissue is highly specialized for this non-shivering thermogenesis.

Non-shivering thermogenesis is regulated mainly by thyroid hormone and the sympathetic nervous system.

Non-shivering thermogenesis is the primary means of heat generation in hibernating mammals and in human infants.

Sellers earned himself an international research reputation mainly through his work on cold adaptation, non-shivering thermogenesis and thyroid function.

They rely on non-shivering thermogenesis.

Merritt et al. (1995) found that its mass declined by half from summer to winter and non-shivering heat production, energy conservation, and reduced body mass.

Some bats are true hibernators and rely upon a rapid, non-shivering thermogenesis of their brown fat deposit to bring them out of hibernation.

Thermogenin is primarily found in brown adipose tissue, or brown fat, and is responsible for non-shivering thermogenesis.

The process, called non-shivering thermogenesis, may involve a substance called brown fat that adults carry in certain areas, like the upper back and side of the neck.

Non-shivering thermogenesis occurs in brown adipose tissue (brown fat) that is present in all eutherians (swine being the only exception currently known).

In infants, the autonomic nervous system may also activate brown adipose tissue to produce heat (non-exercise-associated thermogenesis, also known as non-shivering thermogenesis).

Both are inhibitory to areas in the brainstem which activate non-shivering thermogenesis via brown adipose tissue (BAT) in response to cutaneous cold or prostaglandin E2.

Non-shivering thermogenesis is used by Columba livia, when exposed to cold to generate heat; an increase in Na/K-ATPase activity drives this mechanism in the liver.

They can seek out warm places, fluff up their fur or feathers, reduce skin blood flow, fidget, shiver or indulge in non-shivering thermogenesis by increasing their resting metabolic rate.

There is another technique known as metabolic thermogenesis, which is a non-shivering method of producing adrenalin done with the aid of food, in what is known as calorigenic action.

This is also known as non-shivering thermogenesis, which metabolizes fat but dissipates heat from the proton motive force in mitochondria rather than using oxidative phosphorylation to produce ATP.

It is used to generate heat by non-shivering thermogenesis, and makes a quantitatively important contribution to countering heat loss in neonates which would otherwise occur due to the high surface area-volume ratio.

Fever signals sent to the DMH and rRPa lead to stimulation of the sympathetic output system, which evokes non-shivering thermogenesis to produce body heat and skin vasoconstriction to decrease heat loss from the body surface.

These results suggest a nonshivering component to heat production during acute cold exposure, which can be blocked with propranolol.

Key words: nonshivering thermogenesis, brown fat, 5′-deiodinase, Phodopus sungorus.

No such decrease occurs in liver or kidney, tissues in which nonshivering thermogenesis does not occur.

Key words: nonshivering thermogenesis, propranolol, β-adrenergic blockade, body temperature, exercise, shivering.

These results suggest that repetitive immobilization stress, but not cold acclimation, could enhance nonshivering thermogenesis, at least in part, through an improvement in the responsiveness of adrenocortical hormone secretion to acute stress or cold.

To evaluate the role of adrenocortical hormones in stress- or cold-induced nonshivering thermogenesis, plasma corticosterone (CS) and deoxycorticosterone (DOCS) were measured with the aid of HPLC under various conditions.

These results suggest that heat exposure for 14 days, regardless of the pattern, shifts the lower critical temperature to a high level, and the changes are attributed to an upward shift in the ambient temperature at which nonshivering thermogenesis occurs.

These findings argue against the hypothesis that the local direction of heat from the interscapular brown adipose tissue via Sulzer's vein to the spinal cord has importance in determining the capacity of the rat to respond to noradrenaline or to use nonshivering thermogenesis.

At 20 °C ambient temperature, the expected increase in BAT blood flow associated with nonshivering thermogenesis was observed in control rats, but in fenfluramine-treated rats the increase in BAT blood flow was severely attenuated, and and body temperature were reduced.