ototoxic

However, taking antioxidants helps prevent ototoxic hearing loss, at least to a degree.

Combined with noise, these ototoxic chemicals have an additive effect on a person's hearing loss.

The following list provides an accurate catalogue of ototoxic chemicals:

Ototoxic chemicals interact with mechanical stresses on the hair cells of the cochlea in different ways.

Ototoxic effects are also seen with quinine and heavy metals such as mercury and lead.

For some ototoxic chemical exposures, particularly styrene, the risk of hearing loss can be higher than being exposed to noise alone.

Both furosemide and salicylic acid are considered ototoxic at certain doses.

Ornidyl may interact with bone marrow depressants and ototoxic medications.

When ingested, salicylic acid has a possible ototoxic effect by inhibiting prestin.

Macrolide antibiotics, including erythromycin, are associated with reversible ototoxic effects.

If ototoxic medications must be administered, close attention by the physician to prescription details, such as dose and dosage interval, can reduce the damage done.

Gentamicin is also ototoxic and nephrotoxic, with this toxicity remaining a major problem in clinical use.

Hair cells may also be destroyed chemically by an ototoxic medication, or simply damaged over time by excessively loud noises.

Damage to the cochlea can occur in several ways, for example by viral infection, exposure to ototoxic chemicals, and intense noise exposure.

Due to the difference in the function of these structures, ototoxic poisoning affects the patient differently than in the case of cochlear damage.

Some environmental factors, such as ototoxic medication and noise exposure, appear to be more detrimental to high frequency sensitivity than to that of mid or low frequencies.

Contact with ototoxic chemicals such as styrene, toluene and carbon disulfide heighten the risk of auditory damages.

In some cases, the loss is extremely sudden and can be traced to specific diseases, such as meningitis, or to ototoxic medications, such as Gentamicin.

The most common causes of damage to the vestibular nerve are exposure to ototoxic antibiotics, Ménière's disease, encephalitis and some rare autoimmune disorders.

Ototoxic drugs are drugs which affect the cochlea through the use of a toxin like aminoglycoside antibiotics, which poison hair cells.

These acute ototoxic side effects inhibit ion transport resulting in accumulation of ions in the extracellular space leading to edema.

When a medication causes an ototoxic reaction in either the vestibule or semi-circular canals, the patient suffers from problems in balance and orientation rather than hearing issues.

Specifically, the active amplification function of the outer hair cells is very sensitive to damage from exposure to trauma from overly-loud sounds or to certain ototoxic drugs.

A number of nonsteroidal anti-inflammatory drugs (NSAIDS) have also been shown to be ototoxic.

Like other aminoglycosides, tobramycin is ototoxic: it can cause hearing loss, or a loss of equilibrioception, or both in genetically susceptible individuals.