overexpression

Expression is often done to a very high level and therefore referred to as overexpression.

In general, this leads to an overexpression of the genes.

Overexpression of miR-181b may play an important role in malignant transformation.

Overexpression of CKα-1 has been found to be associated with cancer.

Labels have been applied to the differences in overexpression.

Overexpression of Pom1 can also lead to the formation of new growth ends.

Overexpression is often a characteristic of cancer cell lines.

The mechanism for overexpression of BP1 in breast cancer is unknown.

Additionally, overexpression of Skp2 is correlated with a poor prognosis in breast cancer.

Overexpression of this gene has been shown to increase tumor growth and angiogenesis.

A translocation between chromosome 14 and 18 results in the overexpression of the bcl-2 gene.

Overexpression of p202 in cells decreased the p53 mediated apoptosis.

Loss of function and overexpression experiments on H19 have revealed two things:

Misincorporation may be especially pronounced under conditions of forced and massive gene overexpression.

Overexpression and mutation of the p53 gene is now well described in large bowel cancer.

The gene overexpression may play key roles in carcinogenesis.

Overexpression of β-catenin has been associated with many cancers, including endometrial cancer.

As it turns out, 25 percent of breast-cancer patients exhibit a unique genetic variation called Her2 overexpression.

Thus, the retrovirus allows 20 to 30-fold overexpression of the transfected receptor.

Overexpression of this gene product may be associated with neoplastic transformation for some tumors.

Its amplification and overexpression is especially pronounced in mantle cell lymphomas.

Overexpression has been used in other conditioning paradigms and has produced a mixed set of results.

CD117 is a proto-oncogene, meaning that overexpression or mutations of this protein can lead to cancer.

Plk1 is considered a proto-oncogene, whose overexpression is often observed in tumor cells.

Overexpression of osteonectin is reported in many human cancers such as breast, prostate and colon.