reuptake

The first primary sequence of a reuptake protein was published in 1990.

For this reason, they have largely been replaced by newer selective reuptake drugs.

Cocaine blocks reuptake of dopamine and therefore increases its effects.

D-161 is a chemical known in research circles as a triple reuptake inhibitor.

Ethanol also appears to block the reuptake of adenosine, additionally.

It is also considered a less potent inhibitor of dopamine reuptake.

CP-39,332 is a drug which acts as a serotonin-norepinephrine reuptake inhibitor.

Many drugs interfere with the reuptake of a neurotransmitter from synapses.

On the other hand, selective serotonin reuptake inhibitors are not useful.

Signal termination is a result of reuptake and degradation.

There are several parts to the phrase endocannabinoid reuptake inhibitor.

Some of the most commonly prescribed antidepressants are called reuptake inhibitors.

As the name implies, they block the reuptake of both serotonin and norepinephrine.

This provides a very strong and proportional effect on the blockade of serotonin reuptake.

Diphenhydramine has also been shown to inhibit the reuptake of serotonin.

It is thought to be a reuptake inhibitor.

A recent case report suggests selective serotonin reuptake inhibitors may be helpful.

Some antidepressants work by blocking the reuptake of norepinephrine.

But while the reuptake mechanism may respond quickly, the serotonin system can take weeks or months to readjust.

It acts by inhibiting the reuptake of serotonin and norepinephrine.

The drug also acts as a dopamine reuptake inhibitor.

Astrocytes seem to utilize reuptake mechanisms for a neuroprotective role.

The norepinephrine reuptake inhibitor effects may also play a role in reducing dependence.

The main mechanism of action was instead inhibition of dopamine reuptake.

Troparil is used in scientific research into the dopamine reuptake transporter.