sister chromatid

Diethylaniline may be genotoxic because it has been found to increase the rate of sister chromatid exchange.

One of these cohesin proteins crucial for sister chromatid cohesion is Scc1.

The cohesin multiprotein complex is required for sister chromatid cohesion.

Cohesin subunits bind to each sister chromatid and form a bridge between the two.

Progression from metaphase to anaphase is marked by sister chromatid separation.

As a matter of convention, each sister chromatid is now considered a chromosome, so they are renamed to daughter chromosomes.

Another possibility involves the creation of a dimer where each ring surrounds one sister chromatid.

During mitosis, each sister chromatid forming the complete chromosome has its own kinetochore.

Sister chromatid exchange has also been observed more frequently in B51(+) Behçet's disease.

Defects in the establishment of sister chromatid cohesion have serious consequences for the cell and are therefore tied to many human diseases.

In the fishing pole analogy, the kinetochore would be the "hook" that catches a sister chromatid or "fish".

The staining revealed that few segments were passed to the sister chromatid which were not dyed.

Their results indicate that interfering with this positive securin-separase-cdc14 loop decreases sister chromatid separation synchrony.

In vertebrates, sister chromatid cohesion is released in 2 steps via distinct mechanisms.

These four proteins are essential in yeast, and a mutation in any of them will produce premature sister chromatid separation.

Sister chromatid cohesion is essential for the correct distribution of genetic information between daughter cells and the repair of damaged chromosomes.

Sister chromatid cohesion refers to the process by which sister chromatids are paired and held together during certain phases of the cell cycle.

In this case, the 3' end is degraded and the longer 5' end invades the contiguous sister chromatid, forming a replication bubble.

Changes in patterns of sister chromatid cohesion have been observed in cases of DNA damage.

This disorder shows increased chromosomal breakage and is diagnosed by increased sister chromatid exchanges on chromosomal analysis.

Each sister chromatid is not considered a chromosome in itself, and a chromosome does not always contain two sister chromatids.

If possible, cells use the unmodified complementary strand of the DNA or the sister chromatid as a template to recover the original information.

Sororin is required for stable binding of cohesin to chromatin and for sister chromatid cohesion in interphase.

Diseases arising from defects in cohesin or other proteins involved in sister chromatid cohesion are referred to as cohesinopathies.

Firstly, it targets securin for destruction, enabling the eventual destruction of cohesin and thus sister chromatid separation.