trigger zone

However, there is no symmetrical distribution found between trigger zones and sites of referred sensation.

A scratch on the trigger zone creates a referred sensation in a distant body part but not vice versa.

Radiation to the brain is believed to stimulate the brain's vomiting center or chemoreceptor trigger zone.

This supports the finding that trigger zones in the chest area lead to referred sensation in the legs.

It may have effects directly on the labyrinthine apparatus and on the chemoreceptor trigger zone.

Nausea and vomiting are largely determined by activity in a brainstem area known as the chemoreceptor trigger zone.

These cymbal pads are called three-way cymbals because of their three trigger zones (bell, bow and edge).

Receptors on the floor of the fourth ventricle of the brain represent the chemoreceptor trigger zone.

This activates 5HT3 receptors in the chemoreceptor trigger zone that stimulate vomiting.

However, it is known that the area postrema is the locus, or site, of the chemoreceptor trigger zone for vomiting.

The piriform cortex contains a critical, functionally defined epileptogenic trigger zone, "Area Tempestas".

At very high doses, piribedil has an emetic action on the chemoreceptor trigger zone (CTZ).

A chemoreceptor trigger zone (CTZ).

The drug depresses labyrinth excitability and vestibular stimulation, and it may affect the medullary chemoreceptor trigger zone.

Many TN and ATN patients have pain that is "triggered" by light touch on shifting trigger zones.

Interest also developed in the potential for MNTX to act at the chemoreceptor trigger zone and block the emetic effect of opioids.

The antiemetic properties of domperidone are related to its dopamine receptor blocking activity at both the chemoreceptor trigger zone and at the gastric level.

This is due to the physiology of the CNS vomiting centre and its inputs from the chemoreceptor trigger zone versus the inner ear.

In this indication it blocks the chemoreceptor trigger zone (CTZ) in the brain, which is responsible for causing severe nausea and vomiting.

They both act locally by irritating the gastric mucosa and centrally by stimulating the medullary chemoreceptor trigger zone to induce vomiting.

The chemoreceptor trigger zone is an area of the medulla in the brain that receives inputs from blood-borne drugs or hormones, and communicates with the vomiting center.

Although the specific mechanism through which trimethobenzamide functions is unknown, it is believed to affect the chemoreceptor trigger zone (CTZ) of the medulla oblongata.

One of the ways the chemoreceptor trigger zone implements its effects on the vomiting center is by activation of the opioid mu receptors and delta receptors.

Olanzapine may display an antiemetic effect due to its blockade of several receptors (specifically acetylcholine, dopamine, histamine, and serotonin 5-HT) within the chemoreceptor trigger zone.

Ondansetron reduces the activity of the vagus nerve, which deactivates the vomiting center in the medulla oblongata, and also blocks serotonin receptors in the chemoreceptor trigger zone.