viremia

The virus isolation in cell culture is effective during viremia.

Once control was achieved, the majority of the patients experienced a subsequent rise in viremia.

The antibody reaction plays an important role in decreasing levels of viremia.

Vaccination must be done before secondary viremia takes place for the individual to avoid brain damage or death.

Antibodies can be important in controlling viremia and limiting the severity of infection.

Whatever the route, maternal viremia seems a likely prerequisite for transplacental infection.

Subsequent replication occurs in other organs leading to viremia.

Stage Four: The main point in the infection- where the virus can take over the body's immune system and cause viremia.

Primary viremia refers to the initial spread of virus in the blood from the first site of infection.

Local virus replication will result in viremia, spreading the infection into other tissues and organs.

Known as viremia, the presence of virus in the bloodstream enables it to be widely distributed throughout the body.

Time course of viremia and antibody seroconversion following human immunodeficiency virus exposure.

This factor was no longer predictive when the period of longest control of viremia was considered.

With West Nile virus infection, the viremia usually is transient and people recover very quickly.

When bacteria or viruses are present in the bloodstream, the condition is known as bacteremia or viremia.

Upon infection of the CNS, secondary viremia results and symptoms usually begin.

This sustained replication causes a major viremia, and leads to the development of minor influenza-like symptoms.

Once a human is bitten by the infected mosquito, the virus can gain entry into the bloodstream, causing viremia.

All theories require primary viremia.

Active viremia is caused by the replication of viruses which results in viruses being introduced into the bloodstream.

Likewise, the terms viremia and fungemia simply refer to viruses and fungi in the bloodstream.

Circulating immune complexes, overwhelming viremia, or an uncontrolled immune response may be contributing factors.

Passive viremia is the introduction of viruses in the bloodstream without the need of active viral replication.